By Icon Health Publications
This can be a 3-in-1 reference booklet. It supplies a whole scientific dictionary protecting 1000s of phrases and expressions on the subject of aldosterone. It additionally supplies wide lists of bibliographic citations. eventually, it offers info to clients on the way to replace their wisdom utilizing a variety of web assets. The publication is designed for physicians, clinical scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to familiarize yourself with learn devoted to aldosterone. in the event that your time is effective, this publication is for you. First, you won't waste time looking out the net whereas lacking loads of correct info. moment, the booklet additionally saves you time indexing and defining entries. ultimately, you won't waste time and cash printing 1000's of websites.
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Additional resources for Aldosterone - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Previous work found that Nedd4 and Nedd4-2 reduce ENaC surface expression by targeting the channel for degradation. A defect in this pathway is responsible for Liddle's syndrome. Conversely, serum and glucocorticoidregulated kinase (SGK), a down-stream mediator of aldosterone, increases ENaC surface expression. Our preliminary studies suggest the novel hypothesis that these two pathways are not independent, but that they converge in a common pathway to regulate Na+ absorption. The goal of this project is to test the hypothesis that SGK modulates ENaC surface expression in part through Nedd4-2 binding and phosphorylation, and to understand the mechanisms involved.
We will also test for a synergistic effect of spironolactone with amiloride to lower blood pressure. In Specific Aim #2, we will test the hypothesis that the mechanism whereby the anti-ENaC drugs lower blood pressure is by reducing an intrinsically higher level of ENaC function, as evidenced by lower levels of renin and aldosterone at baseline and greater increments during treatment in responders. In Specific Aim #3, we will test the hypothesis that blood pressure responses to spironolactone are related to the presence of molecular variants in ENaC subunits, variants that have been shown previously to associate with level of blood pressure.
Medicine; University of Illinois at Chicago 1737 West Polk Street Chicago, Il 60612 Timing: Fiscal Year 2004; Project Start 15-JAN-2004; Project End 31-DEC-2005 Summary: (provided by applicant): Dietary salt has a major impact on blood pressure in many patients and is considered to be an important environmental risk factor for hypertension. Understanding the genetic underpinnings causing hypertensin is a necessary step for developing novel and customized therapeutic strategies for hypertension.